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Insulin Resistance: What It Is, Causes, Symptoms & Treatment
2024年11月21日 · Insulin resistance is a complex condition in which your body doesn’t respond as it should to insulin. Insulin is a hormone your pancreas makes that’s essential for regulating blood sugar levels. Several genetic and lifestyle factors can contribute to insulin resistance.
Insulin Resistance: Symptoms, Causes, Tests, and Treatment
2024年4月22日 · Insulin resistance is when cells in your muscles, fat, and liver don’t respond well to insulin, a hormone made by your pancreas that helps manage your blood glucose (blood sugar). Sometimes ...
About Insulin Resistance and Type 2 Diabetes - CDC
2024年5月15日 · What is insulin resistance? Sometimes this finely tuned system can get out of balance. When your body is exposed to too much blood sugar over an extended period of time, you can develop insulin resistance. Here's what that means: A …
Insulin Resistance and Diabetes | ADA
People with insulin resistance, also known as impaired insulin sensitivity, have built up a tolerance to insulin, making the hormone less effective. As a result, more insulin is needed to persuade fat and muscle cells to take up glucose and the liver to continue to store it.
Insulin Resistance: Testing, Symptoms, Diet, Reversal
2024年1月13日 · Insulin resistance occurs when a person's body becomes tolerant to the effects of insulin, a hormone that helps blood sugar enter cells for energy. The exact cause of insulin resistance is unknown but strongly linked to excess body weight and a sedentary lifestyle.
Insulin resistance - Wikipedia
Insulin resistance (IR) is a pathological condition in which cells in insulin-sensitive tissues in the body fail to respond normally to the hormone insulin or downregulate insulin receptors in response to hyperinsulinemia.
Insulin Resistance - StatPearls - NCBI Bookshelf
2020年9月9日 · Insulin resistance, identified as an impaired biologic response to insulin stimulation of target tissues, primarily involves liver, muscle, and adipose tissue. Insulin resistance impairs glucose disposal, resulting in a compensatory increase in beta-cell insulin production and hyperinsulinemia.
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