HL-60

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1.
Conclusions VPA could inhibit the proliferation and induce apoptosis of HL-60 cells in a time-dose dependent manner.
结论VPA对HL-60细胞具有增殖抑制和诱导细胞凋亡作用，且呈剂量-时间依赖性；
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2.
Results The binding activity of these retinoids was well correlated to their ability to induce differentiation of HL-60 cell line.
结果这类化合物的维甲酸受体结合能力与其诱导HL-60细胞成熟分化能力成正相关。
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3.
Objective: To explore the mechanism of hypermethylation pattern of the calcitonin gene in the HL-60 cells.
目的：探讨HL-60细胞降钙素基因高甲基化的发生机制。
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4.
Objective To investigate the telomerase activity in HL-60 cells treated by Astragalus Polysaccharide.
目的研究黄芪多糖对人早幼白血病HL-60细胞端粒酶活性的作用。
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5.
It has been proved that caspases play an important role in irradiation-induced apoptosis of U937 and HL-60 cells.
研究证明，在辐射诱导U937、HL-60细胞凋亡中，半胱氨酸蛋白酶caspases发挥主要作用。
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6.
Conclution: Rapamycin inhibited the proliferation of leukemia HL-60 cells dependent on density and time.
结论：雷帕霉素具有浓度和时间依赖性抑制白血病细胞HL-60增殖。
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7.
The recombinant plasmid was transfected into the cell line HL-60 by electroporation.
采用电转染方法将重组体导入HL-60细胞中；
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8.
Methods: The inhibitation of HL-60 cells by Rapamycin was measured by MTT assay.
方法：用MTT比色法测定雷帕霉素对HL-60细胞增殖的影响；
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9.
Studies with mast cells, HL-60 human leukemic cells and purified G-proteins.
与肥大细胞研究的HL-60人白血病细胞和纯化G蛋白。
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10.
Results: The extracts of hedyotis diffusa can inhibit the HL-60 cell markedly in a dose and time-dependent manner.
结果：白花蛇舌草提取物对HL-60细胞有明显的抑制作用，并且呈剂量-时间依赖关系。
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